Essential Fatty Acid Conspiracies

CountFishula Fish oils (and other highly Polyunsaturated oils), rather than sugar, have been experimentally demonstrated to promote symptoms of type II diabetes.

“Omega-3 fatty acid treatment in type II diabetes leads to rapid but reversible metabolic deterioration…”

“Average blood glucose concentrations during the third week were significantly higher…on PUFA (polyunsaturated fatty acids) than on the saturated fat diet”

“…fish oil reduced the rate of plasma glucose disappearance by 26 %”

In some studies, rather than worsening symptoms, high sucrose diets have been shown not to have an adverse effect on blood sugar, and so called ‘essential fatty acid deficiency’ diets have been exhibited to ‘increase glucose uptake…’.

“Caution should be used when recommending omega-3 fatty acids in type II diabetic persons.”

Avoiding consumption of fish oil, as well as other polyunsaturated fats, is a rational approach to protection from type II diabetes.

Sufficient ingestion of sugar can be protective, in part by preventing the release of stored polyunsaturated fats into the blood, as well as via the suppression of many stress related substances, and the production of some protective and anti-inflammatory fats, including the saturated and omega-9 fats.

The unsaturated fats found in the fish oils, break down quickly and easily into chemicals which have a variety of toxic effects, interfering with metabolism as well as directly preventing cells from being able to use glucose for fuel.

Due to their highly oxidizable nature, these fats have been shown to speedily suppress immune function, often resulting in a temporary reduction in existing processes of inflammation, and the illusory appearance of improvement, as a consequence of some short term symptom reduction.

In the longer term, however, the immunosuppressive impact of these so called ‘anti-inflammatory’ fats have many disease promoting and toxic effects, such as the worsening of the symptoms of diabetes (and issues of blood sugar dysregulation generally), as well as increasing the likelihood of other degenerative and inflammatory conditions, including cancer and Alzheimer’s disease.

Although the saturated fats released into the bloodstream (either from storage in the tissue or from the consumption of fatty food), can temporarily interfere with the ability of the cell to oxidize sugar, they are however, far more stable as well as being unlikely to breakdown into toxic substances, thus allowing the cell to easily return to the proper use of sugar for fuel when it again becomes available.

When sugar is restricted, and when the ability of the cell to oxidize glucose has been impeded (due in large part to the by-products of the breakdown of fish oils and other polyunsaturated fats), many inflammatory substances are released, including cortisol and adrenalin, which further promote the release of the polyunsaturated free fatty acids from storage.

This again encourages the progression of diabetes related issues, worsening hyperglycemia, leading to misconceived ideas regarding the cause of these problems, increasingly popularly placing the blame onto sugar.

One possible therapeutic approach involves experimentation with the gradual increase in use of white sugar and honey in combination with the minerals necessary for a properly functioning metabolism, from sources such as sweet ripe juicy fruits and fruit juice, whilst at the same time avoiding as much as possible, the consumption of the polyunsaturated fish and seed oils.

Such a diet can, in the context of sufficient consumption of protein and nutrients from milk and cheese and gelatin or gelatinous meat, potentially help to prevent the excessive release of polyunsaturated fats held in storage, whilst at the same time promoting the endogenous production of anti inflammatory fats.  The potential increase in production of the saturated and omega-9 fats can further assist in protecting against the by-products of the breakdown of fish oil and other Pufa, as well as improving the ability of the cells to come back to the proper utilization of glucose for fuel, allowing the body to safely and slowly rid itself of the toxic substances via safer means.

See more here

Adverse metabolic effect of omega-3 fatty acids in non-insulin-dependent diabetes mellitus

Dietary supplementation with n-3 fatty acids may impair glucose homeostasis in patients with non-insulin-dependent diabetes mellitus

Polyunsaturated fatty acids may impair blood glucose control in type 2 diabetic patients

Fish-oil supplementation reduces stimulation of plasma glucose fluxes during exercise in untrained males

Effects of Fish Oil Supplementation on Glucose and Lipid Metabolism in NIDDM

Free fatty acids and insulin resistance.

Adipose tissue metabolism in essential fatty acid deficienty. Effects of prostaglandin e1, epinephrine, and ACTH

Metabolic effects of dietary sucrose in type II diabetic subjects

Diabetogenic impact of long-chain omega-3 fatty acids on pancreatic beta-cell function and the regulation of endogenous glucose production.


Image: Allcinema: “Vampire Fish River Monsters”
Artist: Unknown

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