Surviving The War On Cancer.

WonderBird “Has anyone ever heard of a radiologist or surgeon who measured estrogen or the various mediators of inflammation before, during, and after their treatments? Long range survival after breast cancer surgery is affected by the time in the menstrual cycle when the surgery is done.”Ray Peat Phd

“In 3 of 4 reports from major cancer treatment centres…risk of recurrent cancer and/or death increased 5 to 6-fold after 10 years for women receiving surgery during d7-14 of their cycle, compared to those resected during d21-36.”

“Improvement in prognosis was greatest for patients with the highest risk of recurrence…”

Significant improvements in survival were seen in patients receiving surgical treatment during the luteal phase of the cycle – with increased plasma progesterone levels – compared to those with the highest estrogen levels.

“We conclude that accurate menstrual histories should be included in the medical record from now on for all premenopausal women receiving any surgical procedure upon the breast…”

Although some studies disagree with the extent to which long term survival rates – for breast cancer surgery patients – are impacted upon by ‘menstrual timing’, there appears to be enough evidence to warrant further investigation. Until otherwise proven, breast cancer surgical procedures should be scheduled to take place during the luteal phase of the cycle, when progesterone levels are at their highest relative to estrogen.

Menstrual timing – apart from being significant in relation to long term breast cancer surgery outcomes – is also relevant as a clue to the important impact of comparative levels of estrogen to progesterone (in the body) on the promotion and development of many types of cancer. Also, it provides additional insight with regards to the potential effectiveness of non surgical treatments such as the use of progesterone (and other safe and effective substances) for breast cancer and cancer in general.

The estrogen suppressing – and most likely progesterone encouraging – effects of aspirin, cyproheptadine and vitamin A (Retinol) have also been demonstrated to be effective against breast cancer. Other safe substances such as testosterone (combined with aromatase inhibition), niacinamide and caffeine have shown good results.

This all makes sense in the context of a physiologically honest and rational explanation regarding the role played by estrogen and progesterone (as well as a variety of inflammatory and stress related substances) in relation to proper metabolic performance and function.

The same rationale can be used to justify the avoidance of unnecessary exposure to radiation and excessive use of the polyunsaturated fats, as well as experimentation with a metabolism enhancing (and stress protective) diet including protein and nutrients from milk and cheese and gelatin, and plenty of sugar from sweet ripe fruit, fruit juice, white sugar and honey.

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Timing of breast cancer surgery during the luteal menstrual phase may improve prognosis.

Timing of surgery for breast cancer.

Timing of surgery in relation to the menstrual cycle in premenopausal women with operable breast cancer.

Effects on survival of menstrual cycle phase of adjuvant surgical oophorectomy in premenopausal women with breast cancer.

Menstrual timing of treatment for breast cancer.

Progesterone receptor modulates ERĪ± action in breast cancer.


Aspirin blocks growth of breast tumor cells and tumor-initiating cells and induces reprogramming factors of mesenchymal to epithelial transition.

Deciphering the divergent roles of progestogens in breast cancer

Identification of Cyproheptadine as an Inhibitor of SET Domain Containing Lysine Methyltransferase 7/9 (Set7/9) That Regulates Estrogen-Dependent Transcription.

Rapid response of breast cancer to neoadjuvant intramammary testosterone-anastrozole therapy: neoadjuvant hormone therapy in breast cancer

Retinol inhibits aromatase activity and expression in vitro.

The inhibitory effect of caffeine on hormone-induced rat breast cancer.

Mitochondrial complex I activity and NAD+/NADH balance regulate breast cancer progression

Genomic agonism and phenotypic antagonism between estrogen and progesterone receptors in breast cancer

Possible net harms of breast cancer screening: updated modelling of Forrest report

FoxA and LIPG endothelial lipase control the uptake of extracellular lipids for breast cancer growth

The contribution of cytotoxic chemotherapy to 5-year survival in adult malignancies.

Regular and low-dose aspirin, other non-steroidal anti-inflammatory medications and prospective risk of HER2-defined breast cancer: the California Teachers Study


Image: Pia Guerra: “A message to SCOTUS and Hobby Lobby from WW”

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