Surviving The War On Cancer.
“Has anyone ever heard of a radiologist or surgeon who measured estrogen or the various mediators of inflammation before, during, and after their treatments? Long range survival after breast cancer surgery is affected by the time in the menstrual cycle when the surgery is done.” – Ray Peat Phd
“Improvement in prognosis was greatest for patients with the highest risk of recurrence…”
Significant improvements in survival were seen in patients receiving surgical treatment during the luteal phase of the cycle – with increased plasma progesterone levels – compared to those with the highest estrogen levels.
Although some studies disagree with the extent to which long term survival rates – for breast cancer surgery patients – are impacted upon by ‘menstrual timing’, there appears to be enough evidence to warrant further investigation. Until otherwise proven, breast cancer surgical procedures should be scheduled to take place during the luteal phase of the cycle, when progesterone levels are at their highest relative to estrogen.
Menstrual timing – apart from being significant in relation to long term breast cancer surgery outcomes – is also relevant as a clue to the important impact of comparative levels of estrogen to progesterone (in the body) on the promotion and development of many types of cancer. Also, it provides additional insight with regards to the potential effectiveness of non surgical treatments such as the use of progesterone (and other safe and effective substances) for breast cancer and cancer in general.
The estrogen suppressing – and most likely progesterone encouraging – effects of aspirin, cyproheptadine and vitamin A (Retinol) have also been demonstrated to be effective against breast cancer. Other safe substances such as testosterone (combined with aromatase inhibition), niacinamide and caffeine have shown good results.
This all makes sense in the context of a physiologically honest and rational explanation regarding the role played by estrogen and progesterone (as well as a variety of inflammatory and stress related substances) in relation to proper metabolic performance and function.
The same rationale can be used to justify the avoidance of unnecessary exposure to radiation and excessive use of the polyunsaturated fats, as well as experimentation with a metabolism enhancing (and stress protective) diet including protein and nutrients from milk and cheese and gelatin, and plenty of sugar from sweet ripe fruit, fruit juice, white sugar and honey.
See more here
Timing of breast cancer surgery during the luteal menstrual phase may improve prognosis.
Timing of surgery for breast cancer.
Menstrual timing of treatment for breast cancer.
Progesterone receptor modulates ERĪ± action in breast cancer.
BRCA1 DEFICIENCY EXACERBATES ESTROGEN INDUCED DNA DAMAGE AND GENOMIC INSTABILITY
Deciphering the divergent roles of progestogens in breast cancer
Retinol inhibits aromatase activity and expression in vitro.
The inhibitory effect of caffeine on hormone-induced rat breast cancer.
Mitochondrial complex I activity and NAD+/NADH balance regulate breast cancer progression
Possible net harms of breast cancer screening: updated modelling of Forrest report
FoxA and LIPG endothelial lipase control the uptake of extracellular lipids for breast cancer growth
The contribution of cytotoxic chemotherapy to 5-year survival in adult malignancies.
#estrogenocide
#progestpower
#raypeat
Image: Pia Guerra: “A message to SCOTUS and Hobby Lobby from WW”
