Mutations In The Cancer Gene Connection

CancerAttack “As long as the lump is defined as an alien material, killing it by any means seems reasonable, but if it is seen as the body’s attempt to repair itself, then killing it is no more reasonable than it would be to cut the spots out of someone with smallpox.”Ray Peat Phd

It is a widely held belief that cancer is the product of gene mutations, giving rise to the possibility of tumor development if ‘rogue’ cells are allowed the opportunity to multiply. Removing all such cells – with the use of treatments including surgery, radiation and poisonous drugs – is said to be necessary and effective.

“Breast cancer is the second leading cause of cancer deaths in the United States….Literature has shown that cancer cells lose their tumorigenic potential and display ‘normal’ behavior when placed into ‘normal’….environments.”

Experiments looking at the behaviour of living cells – including cells taken directly from malignant tumors – have shown that it is the surrounding tissue environment which determines whether a cell becomes cancerous or not.

“Here, we show that human…breast cancer cells proliferate and contribute to normal mammary gland development…without tumor formation.”

Estrogen, radiation, and the polyunsaturated fats have all been experimentally shown to promote breast cancer (as well as other cancers), interacting powerfully with each other – as well as with pesticides and other toxic chemicals – to promote inflammation and numerous other factors which interfere with cellular function.

“The results suggest there is significant amount of crosstalk between the tissue microenvironment and cancer cells…”

Maintaining a healthy thyroid metabolism helps to protect against inflammation and the release of many substances – including serotonin and prolactin – which promote the overproduction of estrogen, causing systemic changes in the structure of tissues, which can lead to the development of cancer.

“These results suggest the dominance of a “normal” microenvironment over tumor development and the possible control or “normalization” of cancer cells…by exposure to factors produced by normal tissues.”

Sufficient consumption of sugar helps to suppress cortisol, preventing the release of polyunsaturated fats from storage, once again limiting the tissue disorganising and carcinogenic effects of radiation and estrogen.

“More recently…discovered that in order to become tumorigenic, an oncogene must be in an environment permissive for tumor development…Thus, if conditions did not favor tumorigenesis, no tumor would grow.”

A diet with enough protein – from milk cheese and gelatin – and plenty of sugar – from sweet ripe juicy fruits, fruit juices, white sugar or honey – avoiding intake of polyunsaturated fats from all sources and limiting difficult to digest starches and fibers – from grains, seeds, legumes and under cooked vegetables – can help to improve energy systems and digestive function.

“These results suggest…that cancer cells may be restored to ‘normal’ function in the appropriate tissue microenvironment.”

Another way of saying this is that the promotion of a well functioning thyroid metabolism, helps to minimize the estrogen promoting release of bacterial endotoxin and serotonin (as well as numerous other stress and inflammation promoting substances), and is therefore a reasonable approach to improvement of tissue environments which can enable cells to develop healthily, or return to normal function when conditions improve.

See more here

Human Breast Cancer Cells Are Redirected to Mammary Epithelial Cells upon Interaction with the Regenerating Mammary Gland Microenvironment In-Vivo

BRCA1 deficiency exacerbates estrogen-induced DNA damage and genomic instability.

Estrogen Carcinogenesis in Breast Cancer

Recent Oral Contraceptive Use by Formulation and Breast Cancer Risk among Women 20 to 49 Years of Age

Breast cancer risk in relation to occupations with exposure to carcinogens and endocrine disruptors: a Canadian case–control study


Progesterone receptor modulates ERα action in breast cancer

Genomic agonism and phenotypic antagonism between estrogen and progesterone receptors in breast cancer

The contribution of cytotoxic chemotherapy to 5-year survival in adult malignancies.

The regulation of adenohypophyseal prolactin secretion: effect of triiodothyronine and methylene blue on estrogenized rat adenohypophysis.


Artist: Unknown

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