Aspirin! Because You May Not Need To F%#k Cancer After All.

wedopainkilling There are lots of reasons to be angry about cancer, but when you find out just how effective aspirin is, as both a preventative measure and treatment (even cure), you might move from anger and suffering, skipping past worry and sadness, straight on up to murderous intent.

“Observational studies show that regular use of aspirin reduces the long-term risk of several cancers and the risk of distant metastasis. Results of methodologically rigorous studies are consistent with those obtained from randomised controlled trials…”

“We find that ASA [aspirin] not only prevents breast tumor cell growth…and tumor growth…but also significantly reduces the self-renewal capacity and growth of breast tumor-initiating cells/breast cancer stem cells…and delays the formation of a palpable tumor.”

“The goal in this study was to investigate the antitumor effect of aspirin in glioblastoma cells and the molecular mechanism involved in its antineoplastic activities…results suggest that aspirin is a potent antitumor agent, and that it exerts its antineoplastic action by inhibition of the β-catenin/TCF signaling pathway in glioma cells.”

Of course aspirin isn’t only effective in relation to cancer, because that isn’t how biology and metabolism works, and – contrary to the modern mainstream medical myth – cancer is actually a biological, metabolic issue.

“…aspirin…an anti-inflammatory agent…is currently used extensively as a cardioprotective and antithrombotic agent…regular use of aspirin is associated with a reduced risk for colorectal, oesophageal, breast, lung, prostate, liver and skin cancers…both constituent groups of aspirin…the acetyl and salicylate moieties have distinct targets that…contribute to its anticancer effects.”

“Post-diagnosis use of aspirin in patients with gastrointestinal tract malignancies is associated with increased survival in cancers with different sites of origin and biology.”

“…aspirin inhibits protein aggregation and the ensuing toxicity of aggregates through its acetyl-donating activity. This mechanism may contribute to the neuro-protective, cardio-protective, and life-prolonging effects of aspirin.”

In fact aspirin can be helpful with almost any physiological problem arising out of exposure (both internally and externally) to chronic and acute stress, and stress is a fundamental driver of cancer and many other inflammatory diseases.

“…findings suggest that aspirin use may prevent incident breast, colon, pancreatic, and ovarian cancer in elderly women.”

“…aspirin…appears to reduce staphylococcal virulence…aspirin may have the potential to be an effective adjunctive agent in the treatment of serious hospital- or community-acquired S. aureus infections.”

“Aspirin can reduce oxidative stress and protect against oxidative damage….evidence suggests…beneficial effects of aspirin…in mood disorders and schizophrenia, and…data suggests…aspirin is associated with a reduced risk of AD [Alzheimer’s].”

“Aspirin therapy, in doses commonly employed in practice, has an excellent safety profile in rheumatoid arthritis…Aspirin therapy merits reconsideration as adjunctive therapy for the management of rheumatoid arthritis.”

Aspirin helps to protect against stress by improving the function of metabolism in general. One way it does this, is by protecting against the escalation of inflammation (and numerous other symptoms) arising as a result of interference with thyroid energy systems.

Suppression of thyroid energy metabolism promotes (and is promoted by) inflammation. Inflammation and thyroid dysfunction are interconnected with issues relating to proper cellular and mitochondrial performance, interference with enzyme activity, increased use of fat (and decreased use of sugar) for fuel, combined with more exposure to free fatty acids, lipid peroxidation and oxidative stress. Aspirin has beneficial effects in relation to all of these things, and all of these things have been shown to be involved in the development of cancer and metabolic illness in general.

“Epidemiologic and clinical studies indicate that inflammation is correlated with an increased risk of recurrence in breast cancer patients…multiple meta-analyses showed that patients with cardiovascular diseases treated with…aspirin, have reduced cancer risk.”

“Our observations confirm that low-T3 levels are commonly found in ESRD [end-stage renal disease] patients on PD [Peritoneal dialysis] and show that this alteration is linked to low-grade inflammation and death in this population.”

“Neutrophils have a fundamental role in inflammatory responses…neutrophils specifically support metastatic initiation…neutrophil-derived leukotrienes aid the colonization of distant tissue by selectively expanding the sub-pool of cancer cells that retain high tumorigenic potential…inhibition of the leukotriene-generating enzyme arachidonate 5-lipoxygenase…abrogates neutrophil pro-metastatic activity and consequently reduces metastasis.”

“…we provide new molecular mechanisms underlying the pleiotropic response to ASA by demonstrating that this NSAID promotes the formation of endogenous anti-inflammatory compounds…and modulates 5-LO [5-lipoxygenase] activity…decreasing 5-LO activity in macrophages…a central role in the control of inflammation…”

This is a good way to understand some of the reasons why aspirin is beneficial in relation to protection from heart disease and stroke, but also depression, diabetes and insulin resistance. And then there is its use for the treatment of Alzheimer’s or neuroprotection in general. Not to mention it’s effectiveness in relation to hypertension, pregnancy and fertility issues, and an almost endless list of other symptoms or conditions.

“…neurological disorders, including Alzheimer’s…Huntington’s…and Parkinson’s disease…are characterized by accumulation of insoluble protein aggregates…aspirin inhibits protein aggregation…”

“LDA [low dose aspirin] increased the chances of clinically recognized pregnancy and live birth in women with moderately elevated hsCRP and prior pregnancy loss.”

There is a huge amount of quality scientific experimentation (available for pretty much anybody to look at), which demonstrates the many powerful ways aspirin can protect against disease – including many regarding cancer – and not just simply as a preventative measure.

“We conducted a prospective study from 1992 through 1999 among 28 283 postmenopausal women…There was a trend of decreasing risk of pancreatic cancer incidence with increasing frequency of aspirin use per week.”

“…patients in the study had all stages of cancer at diagnosis—“When you look at the effects of aspirin stratified by cancer stage, you still see the same positive effects.””

“Daily aspirin reduced deaths due to several common cancers during and after the trials. Benefit increased with duration of treatment and was consistent across the different study populations…”

Scientists (working for governments and pharmaceutical companies) all over the world have been attempting to use the physiological effects of aspirin as a model to assist in finding a cure for cancer. But is that really all that they are looking for?

“…a novel, gastrointestinal-safe phosphatidylcholine (PC)-associated aspirin, PL2200 Aspirin, possesses the same or more pronounced actions versus unmodified aspirin with regard to antiplatelet effects…and chemoprevention.”

There seems to be an ongoing race to develop new products modeled on aspirin (which would provide similar positive results in relation to cancer), but different enough to be able to be patented, as a means to making oodles and oodles of dinero.

“…we report a significant reduction in HNC [head and neck cancer] risk with aspirin use, with the strongest protective effect for laryngeal cancers. No association was observed between HNC and ibuprofen use.”

Unfortunately however, there is no guarantee that whatever is produced will match the manner in which aspirin (tried and tested for many decades) works to safely provide physiological protection, including chemotherapeutic benefit. But it’s hard to make the big money out of something so widely and cheaply available, even if it does work better than more recent products.

Many of the substances which are known to rise in times of stress – cortisol, serotonin, estrogen, nitric oxide, lactate, growth hormone, prolactin, the polyunsaturated free fatty acids and prostaglandins – do so because of the ways that stress interferes with metabolic function.

The stress substances are involved in the promotion of inflammatory conditions, and are known to rise under circumstances where thyroid energy metabolism is suppressed. Aspirin has been demonstrated to be able to reduce exposure to the disease promoting stress materials.

“…results suggest that postmenopausal women who regularly use aspirin…may have lower estrogen levels…We observed significant inverse associations between total NSAID use and concentrations of estradiol and free estradiol…analgesics that decrease aromatase activity via suppression of COX expression and prostaglandin synthesis also may decrease estrogen concentrations”

“The inhibition of iNOS expression by aspirin was further associated with a reduced ability…to produce TNF-alpha. This study could provide new mechanisms of action for aspirin in the treatment of the inflammation-related…diseases.”

“The results clearly show that…ASA ingestion significantly blunted the increased serum ACTH, beta-endorphin, cortisol, and GH [growth hormone] levels…and was associated with reduced cortisol concentration…[and] a significantly reduced total PRL [prolactin] response to stress condition[s]…”

“Aspirin (ASA), a platelet aggregation inhibitor, inhibits 5-HT [serotonin] release from platelets…Plasma 5-HT was measured by high-performance liquid chromatography…The effects of ASA was associated with a reduction of 5-HT.”

A lack of energy supply (in combination with continuous exposure to excessive levels of the stress substances and promoters of inflammation) has been demonstrated to be a significant driving force behind the onset and development of cancer. Sugar is an optimal energy source and a fundamental anti-stress substance.

“We show that the targeted therapy with iNOS inhibitors is able to inhibit not only tumor cell proliferation but also…self-renewal and migration, reducing tumor growth, tumor initiation, and the number of lung metastases…”

“5-HT [serotonin] stimulates proliferation of PC cells and 5-HTR1A antagonists inhibit proliferation. Thus, we propose that 5-HT has an important role in tumor progression…”

“17-β-estradiol can act as a carcinogenic agent without the need of the ERα…The knowledge that breast cancer in women is associated with prolonged exposure to high levels of estrogens gives relevance to this model of estrogen induced carcinogenesis.”

“Dietary soy isoflavones increase metastasis to lungs in a model of breast cancer and a recent study reported significantly increased cell proliferation in breast cancer patients who used soy supplementation. The soy isoflavone daidzein is a major food-derived phytoestrogen that is structurally similar to estrogen.”

Even when enough sugar is being provided (and unused supplies are available), ongoing exposure to inflammation and the substances of stress, are important factors which can interfere with the proper utilization of available sugar for energy, and this can make a return to normal function difficult. Aspirin can help protect against the things that interfere with the use of sugar for fuel and stress reduction.

“Insulin-stimulated whole-body glucose uptake was decreased by 37% with the lipid infusion…Salicylate pretreatment prevented these decreases…Salicylate pretreatment also prevented the lipid-induced decreases in insulin-stimulated skeletal muscle glucose metabolism…salicylate pretreatment prevents lipid-induced skeletal muscle insulin resistance…these results provide important new insights into the mechanism of fat-induced insulin resistance…and suggest a potentially novel class of therapeutic agents for type 2 diabetes.”

“…high dose aspirin treatment improved both fasting and postprandial hyperglycemia in patients with type 2 diabetes, an effect that could be attributed to decreased basal rates of hepatic glucose production, enhanced peripheral insulin sensitivity, and decreased insulin clearance.”

“In fasting conditions, the lipolytic activity of adipocytes is stimulated by catecholamines…aspirin has been reported to reduce catecholamine-stimulated lipolysis…In addition…aspirin reduces release of FA [fatty acids] from adipose tissue directly via inhibition of TNFα-induced lipolysis…”

It’s safe to say that aspirin helps to protect against inflammation and disease, by improving the function of metabolic energy systems in a variety of ways which help reduce stress and interference with cellular performance, often the result of excessive exposure to cancer promoting substances such as cortisol, estrogen, nitric oxide and the breakdown products of polyunsaturated fats.

“Since FAs [fatty acids] are essential for cancer cell proliferation, limiting their availability could provide a therapeutic strategy. From the perspective of lipid metabolism, limiting FA availability could be achieved in several ways…”

“Abnormal lipid metabolism is a hallmark of tumorigenesis. Hence, the alterations of metabolism enhance the development of hepatocellular carcinoma (HCC)…Therapeutically, aspirin is potentially available for HCC through controlling abnormal lipid metabolism.”

“…stress…caused brain expression of iNOS, an increase in plasma glutamate and brain TNF-alpha, induction of oxidative indicators in brain and a fall in brain ATP levels….aspirin…inhibited all these effects caused by stress…”

“Arachidonic acid (ARA) is metabolized by cyclooxygenase (COX) and cytochrome P450 to produce proangiogenic metabolites. Specifically, epoxyeicosatrienoic acids (EETs) produced from the P450 pathway are angiogenic, inducing cancer tumor growth.”

“Aspirin is an anti-inflammatory drug, peroxyl radical scavenger, and antioxidant agent that inhibits phospholipases, nitric oxide synthetases, and cyclooxygenase enzymes…Aspirin decreased lipid peroxidation…we demonstrated the potential effects of aspirin on biochemical and neurobehavioral recovery…”

You can see why aspirin is threatening. For starters, if enough people were convinced of its protective ability in relation to cancer, that could damage the popular myth which states that ‘modern science’ is always moving forward in the direction of greater understanding of the causes and treatments of serious illness.

But just think about what’s been known for decades about the benefits of aspirin, and consider the fact that the dangers of aspirin have been shown in many cases to be overblown. There’s no end to the amount of research that can be done, but it won’t change the fact that aspirin already works right now. So what are they waiting for?

“…aspirin suppressed prostate cancer cell invasion by reducing MMP-9 activity and uPA expression through decreasing of IKK-β-mediated NF-κB activation, indicating that the ability of aspirin to inhibit cell invasion might be useful in the chemoprevention of metastatic prostate cancer.”

“…salicylate…exhibit anti-tumor activity against…leukemia…We…tested…other NSAIDs, including acetaminophen and indomethacin…but did not detect any inhibitory activity…salicylate may be useful for treating inflammation, diabetes, neurodegenerative disease, and other pathologies…”

“Postmenopausal women who used ASA had a significantly lower risk of melanoma, and longer duration of ASA use was associated with greater protection.”

Perhaps another fear is that acknowledging the truth about how aspirin works might eventually promote alternative (biologically valid) explanations for cancer development, and that this will cause a flow-on effect, unmasking ineffective and harmful products or treatment methodologies across the board.

I’m not a doctor or health practitioner or a scientist of any sort, and I’m not here to give advice. Read the arguments. Have a look though the science.

There are however, those who believe that it makes more sense to look at cancer (rather than simply as some kind of genetic disease) as a reasonably normal reaction of cells – doing the best they can to keep you alive and functioning – after a certain amount of time being subjected to ‘unnatural’ (or excessively stressful) conditions.

At moments like this it seems more appropriate – rather than saying ‘f%#k cancer’ – to say ‘f&$k the cancer industry’, and all the related organizations and industries which have helped muddy the waters surrounding the true causes of (and really effective approaches to protecting against or even treating) cancer, in the name of greed and power.

“The present meta-analysis, which involves approximately 300,000 participants from 16 studies, has confirmed the antineoplastic effects of NSAIDs in multi-cancer metastasis…aspirin was the most predominant NSAID given to patients.”

You see, if everything can cause cancer, and nothing can cure cancer, it becomes easier to get away with selling things that actually cause cancer, and things that really can’t cure cancer, whilst at the same time avoiding competition from the things that actually don’t cause cancer and things that really do cure cancer. Was that clear?

See More Here

Aspirin blocks growth of breast tumor cells and tumor-initiating cells and induces reprogramming factors of mesenchymal to epithelial transition.

Aspirin Suppresses the Acquisition of Chemoresistance in Breast Cancer by Disrupting an NFκB–IL6 Signaling Axis Responsible for the Generation of Cancer Stem Cells

Thyroid Hormones, Oxidative Stress, and Inflammation

Aspirin use and the incidence of breast, colon, ovarian, and pancreatic cancers in elderly women in the Iowa Women’s Health Study

Association between nonsteroidal anti-inflammatory drug use and the incidence of pancreatic cancer.

Association of aspirin and nonaspirin nonsteroidal anti-inflammatory drugs with cancer incidence and mortality.

Effect of aspirin and other NSAIDs on postmenopausal breast cancer incidence by hormone receptor status: results from a prospective cohort study.

Association of aspirin and nonsteroidal anti-inflammatory drug use with breast cancer.

Aspirin inhibits arachidonic acid metabolism via lipoxygenase and cyclo-oxygenase in hamster isolated lungs.

Nonsteroidal anti-inflammatory drugs and risk for ovarian and endometrial cancers in the Iowa Women’s Health Study.

Short-term effects of daily aspirin on cancer incidence, mortality, and non-vascular death: analysis of the time course of risks and benefits in 51 randomised controlled trials.

The influence of aspirin on exercise-induced changes in adrenocorticotrophic hormone (ACTH), cortisol and aldosterone (ALD) concentrations.

Effects of regular aspirin on long-term cancer incidence and metastasis: a systematic comparison of evidence from observational studies versus randomised trials.

Expression of serotonin receptors and role of serotonin in human prostate cancer tissue and cell lines.

Low dose aspirin increases 15-epi-lipoxin A4 levels in diabetic chronic kidney disease patients

Obesity Suppresses Cell-Competition-Mediated Apical Elimination of RasV12-Transformed Cells from Epithelial Tissues

Preconception Low-Dose Aspirin Restores Diminished Pregnancy and Live Birth Rates in Women With Low-Grade Inflammation: A Secondary Analysis of a Randomized Trial

Aspirin triggers antiinflammatory 15-epi-lipoxin A4 and inhibits thromboxane in a randomized human trial

Aspirin inhibits both lipid peroxides and thromboxane in preeclamptic placentas.

Changes in thyroid hormones by treatment with aspirin and prednisolone in subacute thyroiditis with hyperthyroidism.

Aspirin and cancer risk: a quantitative review to 2011.

Aspirin, nonaspirin nonsteroidal anti-inflammatory drug, and acetaminophen use and risk of invasive epithelial ovarian cancer: a pooled analysis in the Ovarian Cancer Association Consortium.

Mechanism by which high-dose aspirin improves glucose metabolism in type 2 diabetes

Aspirin reduces hypertriglyceridemia by lowering VLDL-triglyceride production in mice fed a high-fat diet

Neutrophils support lung colonization of metastasis-initiating breast cancer cells

Role of mitochondria in aspirin-induced apoptosis in human gastric epithelial cells

Nonsteroidal anti-inflammatory drugs and risk of ovarian cancer: systematic review and meta-analysis of observational studies.

Aspirin might reduce the incidence of pancreatic cancer: A meta-analysis of observational studies.

Aspirin-Mediated Acetylation Protects Against Multiple Neurodegenerative Pathologies by Impeding Protein Aggregation.

Inhibition of iNOS as a novel effective targeted therapy against triple-negative breast cancer

The epigenetic effects of aspirin: the modification of histone H3 lysine 27 acetylation in the prevention of colon carcinogenesis in azoxymethane- and dextran sulfate sodium-treated CF-1 mice

Personalizing Aspirin Use for Targeted Breast Cancer Chemoprevention in Postmenopausal Women.

Inhibition of arachidonate 5-lipoxygenase triggers massive apoptosis in human prostate cancer cells


Aspirin inhibits the proliferation of hepatoma cells through controlling GLUT1-mediated glucose metabolism

Aspirin attenuates pulmonary arterial hypertension in rats by reducing plasma 5-hydroxytryptamine levels.

Aspirin inhibits LPS-induced macrophage activation via the NF-κB pathway

Effect of daily aspirin on long-term risk of death due to cancer: analysis of individual patient data from randomised trials.

Cyclooxygenase-2 polymorphisms, aspirin treatment, and risk for colorectal adenoma recurrence–data from a randomized clinical trial.

Aspirin induces Nrf2‐mediated transcriptional activation of haem oxygenase‐1 in protection of human melanocytes from H2O2‐induced oxidative stress

Aspirin is associated with lower melanoma risk among postmenopausal Caucasian women: the Women’s Health Initiative.

Subclinical hypothyroidism is linked to micro-inflammation and predicts death in continuous ambulatory peritoneal dialysis

Case–Control Study of Aspirin Use and Risk of Pancreatic Cancer

Aspirin Use and Reduced Risk of Pancreatic Cancer.

Aspirin (ASA) regulates 5-lipoxygenase activity and peroxisome proliferator-activated receptor α-mediated CINC-1 release in rat liver cells: novel actions of lipoxin A4 (LXA4) and ASA-triggered 15-epi-LXA4

Aspirin Inhibits IKK-β-mediated Prostate Cancer Cell Invasion by Targeting Matrix Metalloproteinase-9 and Urokinase-Type Plasminogen Activator

Obesity, Aspirin, and Risk of Colorectal Cancer in Carriers of Hereditary Colorectal Cancer: A Prospective Investigation in the CAPP2 Study

Antioxidant properties of aspirin: characterization of the ability of aspirin to inhibit silica-induced lipid peroxidation, DNA damage, NF-kappaB activation, and TNF-alpha production.

Effect of Aspirin on Spinal Cord Injury: An Experimental Study.

Estimates of benefits and harms of prophylactic use of aspirin in the general population

Aspirin, lysine, mifepristone and doxycycline combined can effectively and safely prevent and treat cancer metastasis: prevent seeds from gemmating on soil

Is aspirin use associated with a decreased risk of ovarian cancer? A systematic review and meta-analysis of observational studies with dose-response analysis.

Regular and low-dose aspirin, other non-steroidal anti-inflammatory medications and prospective risk of HER2-defined breast cancer: the California Teachers Study

Aspirin-Mediated Acetylation Protects Against Multiple Neurodegenerative Pathologies by Impeding Protein Aggregation.

Serotonin promotes tumor growth in human hepatocellular cancer.

Aspirin: a review of its neurobiological properties and therapeutic potential for mental illness

A reevaluation of aspirin therapy in rheumatoid arthritis.

Aspirin suppresses the abnormal lipid metabolism in liver cancer cells via disrupting an NFκB-ACSL1 signaling.

Assessment of the Prophylactic Role of Aspirin and/or Clopidogrel on Experimentally Induced Acute Myocardial Infarction in Hypercholesterolemic Rats

Salicylic acid attenuates virulence in endovascular infections by targeting global regulatory pathways in Staphylococcus aureus

Unlocking Aspirin’s Chemopreventive Activity: Role of Irreversibly Inhibiting Platelet Cyclooxygenase-1

Effect of daily aspirin on risk of cancer metastasis: a study of incident cancers during randomised controlled trials

Aspirin inhibits inducible nitric oxide synthase expression and tumour necrosis factor-alpha release by cultured smooth muscle cells.

Risk of prostate cancer in low-dose aspirin users: A retrospective cohort study.

Analgesic medication use and risk of epithelial ovarian cancer in African American women

Non-steroidal anti-inflammatory drug and aspirin use and the risk of head and neck cancer

Aspirin curtails the acetaminophen-induced rise in brain norepinephrine levels.

A Switch from White to Brown Fat Increases Energy Expenditure in Cancer-Associated Cachexia

ARF and p53 coordinate tumor suppression of an oncogenic IFN-β-STAT1-ISG15 signaling axis

Aspirin and salicylates inhibit the IL-4- and IL-13-induced activation of STAT6.

Acetylsalicylic acid inhibits the pituitary response to exercise-related stress in humans.

Aspirin Prophylaxis for Migraine with Aura: An Observational Case Series

Aspirin for the prevention of colorectal cancer

Salicylate or aspirin inhibits the induction of the inducible nitric oxide synthase in rat cardiac fibroblasts.

Aspirin therapy reduces the ability of platelets to promote colon and pancreatic cancer cell proliferation: implications for the oncoprotein c-MYC.

Analgesic use and sex steroid hormone concentrations in postmenopausal women

Aspirin dose dependently inhibits the interleukin-1β–stimulated increase in inducible nitric oxide synthase, nitric oxide, and prostaglandin E2 production in rat ovarian dispersates cultured in vitro

Aspirin is associated with lower melanoma risk among postmenopausal Caucasian women: the Women’s Health Initiative

Salicylate, diflunisal and their metabolites inhibit CBP/p300 and exhibit anticancer activity

2306 Aspirin and gastro intestinal malignancies; improved survival not only in colorectal cancer?

Aspirin Use and Colorectal Cancer Survival According to Tumor CD274 (Programmed Cell Death 1 Ligand 1) Expression Status

Aspirin in the Treatment of Cancer: Reductions in Metastatic Spread and in Mortality: A Systematic Review and Meta-Analyses of Published Studies

Use of Aspirin postdiagnosis improves survival for colon cancer patients

Effect of low-dose aspirin use on survival of patients with gastrointestinal malignancies; an observational study

Aspirin: a review of its neurobiological properties and therapeutic potential for mental illness

Survival Benefits for Low-Dose Aspirin in GI Cancers

Long-Term Effects of Aspirin on Colorectal Cancer

NSAIDs Use and Reduced Metastasis in Cancer Patients: results from a meta-analysis

Aspirin, cyclooxygenase inhibition and colorectal cancer

Antitumor effect of aspirin in glioblastoma cells by modulation of β-catenin/T-cell factor-mediated transcriptional activity.

Salicylate Downregulates 11β-HSD1 Expression in Adipose Tissue in Obese Mice and in Humans, Mediating Insulin Sensitization

Induction of proto-oncogene BRF2 in breast cancer cells by the dietary soybean isoflavone daidzein

Cellular Fatty Acid Metabolism and Cancer

Pharmacologic inhibition of fatty acid oxidation sensitizes human leukemia cells to apoptosis induction

Cyclooxygenase-derived proangiogenic metabolites of epoxyeicosatrienoic acids.

Chronic cellular hypoxia as the prime cause of cancer: what is the de-oxygenating role of adulterated and improper ratios of polyunsaturated fatty acids when incorporated into cell membranes?

Prevention of fat-induced insulin resistance by salicylate

Molecular targets of aspirin and cancer prevention

Evaluation of nootropic and neuroprotective effects of low dose aspirin in rats

Low-dose aspirin and cancer mortality: a meta-analysis of randomized trials.




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