No Need To Trip Over It

BrekkaDelic Even though it’s all the buzz, you don’t need to take LSD or mushrooms, to reduce symptoms of depression and anxiety. There are safer, more reliable ways, to improve or even rid yourself of mental dysregulation issues, and often all it takes is some dietary or lifestyle changes.

In fact, unless you think psychedelic drugs have special magical healing properties, and that you just need to take some and all your problems will miraculously disappear, it would be fair to say that learning how to avoid things that damage metabolic function, increase stress, and inhibit brain function, is a far more reasonable starting point.

You could even consider the possibility of adding into your diet, a few every day ingredients that are known to provide powerful pro-metabolic, anti-stress, anti-inflammatory, brain fueling effects, before you decide to use what some consider to be poorly understood drugs, to do a so called ‘brain reset’.

I mean, let’s assume for a second that hallucinogens can help, it’s not like they’re going to work by making you less metabolically healthy, are they? Surely there are more reliable, less mysterious ways to improve metabolism, that don’t involve summoning evil spirits, or having conversations with aliens.

And call me conspiratorial, but I think there’s good reason to be suspicious of drugs that suddenly start to receive mainstream promotion for the treatment of mental disorders, especially when those same drugs have been officially warned against for decades, because of their ability to cause what they are now saying they can treat. Were they wrong before, or are they wrong now?

What about all the ‘anti-depressants’ that have been pushed for the last few decades because they raise serotonin? I thought they were supposed to be the solution. How does that align with science showing that LSD works by reducing serotonin exposure? And if LSD raises serotonin, like some have tried to argue, aren’t there plenty of drugs that already do that? The truth of the matter is, intentional or not, there is no consistent or coherent reasoning validating any of the main treatment approaches.

Funnily enough though, it doesn’t seem to make a difference, probably because people have been conditioned to accept that whenever the word science gets used, whatever information comes with that, can and must be trusted. Anything else is simply pseudo-scientific woo woo. And granted, lots of ‘alternative’ treatments are.

But what I’m talking about isn’t ‘alternative science’, it’s just science that makes sense, but disagrees with the mainstream narrative. A lot of this uncooperative biological science, fits inside a logical view of how metabolic issues can promote psychological dysfunction, and yet has been set aside as if it doesn’t exist, and most people never even hear about it.

This includes science showing the dangers of the most popular treatment approaches for depression and anxiety, how they can cause what they are supposed to be treating, and how that makes sense in light of the demonstrated causes of many other metabolic illnesses, that often go together with mood related issues. And in any case, it’s pretty obvious that the official ways have not been working very well.

I’m not suggesting that science and nutrition are the solution to every problem effecting the mind, but what we’re talking about here, is some ways to improve basic function and resilience, to raise protection from the stresses of this world, whether biochemical, social, environmental, or spiritual. Not some potion that will ‘reset’ your life with just a few little drops.

I have written articles (with lots of studies attached), showing the things that have been closely associated with psychological dysfunction. Hypothyroidism, inflammation, oxidative stress, rising levels of cortisol, serotonin, nitric oxide, estrogen, bacterial endotoxin and lactate, as well as cholesterol, GABA and dopamine issues, and some other things. All of these can be symptoms (and promoters) of excess stress and metabolic interference.

And all of these can be influenced with simple and safe dietary changes that most people have never tried because they never knew they could, and because they have been propagandized into believing a completely false version of ‘nutritional science’, spread by people who may as well be tripping.

One of the biggest dietary hallucinations is the idea that sugar causes disease, and quitting sugar treats disease. This goes well with the delusional belief that PUFAs are anti-inflammatory and heart-protective. Not to mention fantasies about salt, and iron, and cholesterol, and saturated fats, and dairy, and lots of other figments of the official imagination.

I know every situation isn’t identical, and you can’t just eat a bit of sugar and voila, your problems all go away. But if you have experienced the way something like a ‘stomach bug’ for instance, can effect your emotional state, you have been given a clue as to the dramatic difference a well functioning metabolism can make to the way you feel, even in high stress circumstances.

That’s why it makes sense to first see what happens if you get rid of the main things that can poison metabolism, and start to include the main things that can fuel metabolism (and the brain), and go from there. If you haven’t tried that, then you aren’t in a position to work out what to do after that.

Sometimes, just a bit more sugar and a little less PUFAs can mean slightly less cortisol and adrenaline, and not as much endotoxin, which can then mean lower serotonin, tipping the balance away from excess estrogen, reducing interference with mitochondrial respiration, moving things away from lactate and nitric oxide, improving regulation of dopamine and GABA, and before you know it, your liver works better and you’ve improved thyroid energy system function, and just like magic, you sleep deeper and you feel a bit better.

Other times, it won’t be so straightforward. So don’t take this as a dietary prescription. I’m not a doctor, and it isn’t intended as one. But it’s the idea that’s important. No matter what I say, it can be complicated, and you’re going to have to figure out how to apply all of this to your life and particular circumstances. And like I said, there can be more to it than just a few dietary changes or a few pills. But if you’re being lied to about the effects of food on physiology, it can be an unnecessarily difficult up hill battle, and I don’t believe adding psychedelics into the mix, is the solution.

If you like what I have to say, and you want more information (including lots of studies), showing simple ways to improve metabolic function and reduce mood related issues, please check out some of my other articles, including Are You Running On Empty?. And please share this and sign the email list up top.

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Cyproheptadine Use in Children With Functional Gastrointestinal Disorders

Evidence that brain glucose availability influences exercise-enhanced extracellular 5-HT level in hippocampus: a microdialysis study in exercising rats

Lipid Peroxidation and Depressed Mood in Community-Dwelling Older Men and Women

The use of thyroid hormones in the treatment of depression

Decreasing TSH levels in patients with Seasonal Affective Disorder (SAD) responding to 1 week of bright light therapy

Nitric oxide levels in chronic liver disease patients with and without oesophageal varices

The 5-HT1A Receptor and the Stimulus Effects of LSD in the Rat

Serotonin Synthesis and Reuptake in Social Anxiety Disorder

Indigenous Bacteria from the Gut Microbiota Regulate Host Serotonin Biosynthesis

Role of the 5-HT2A Receptor in Self- and Other-Initiated Social Interaction in Lysergic Acid Diethylamide-Induced States: A Pharmacological fMRI Study

5-HT1A and 5-HT1B receptor agonists and aggression: a pharmacological challenge of the serotonin deficiency hypothesis.

Antagonist properties of d-LSD at 5-hydroxytryptamine2 receptors.

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Modern Clinical Research on LSD

Synthesis of serotonin in traumatized rat brain.

The GABAergic Deficit Hypothesis of Major Depressive Disorder

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Whole-Body Hyperthermia for the Treatment of Major Depressive Disorder

Role of Nitric Oxide in Stress-Induced Anxiety: From Pathophysiology to Therapeutic Target.

Classical hallucinogens as antidepressants? A review of pharmacodynamics and putative clinical roles

Changes in global and thalamic brain connectivity in LSD-induced altered states of consciousness are attributable to the 5-HT2A receptor.

A controlled trial of methylene blue in severe depressive illness.

Lysergic acid diethylamide (LSD) is a partial agonist of D2 dopaminergic receptors and it potentiates dopamine-mediated prolactin secretion in lactotrophs in vitro.

Mobilization of arachidonate and docosahexaenoate by stimulation of the 5-HT2A receptor in rat C6 glioma cells.

Mice Genetically Depleted of Brain Serotonin Do Not Display a Depression-like Behavioral Phenotype

Serotonin-induced decrease in brain ATP, stimulation of brain anaerobic glycolysis and elevation of plasma hemoglobin; the protective action of calmodulin antagonists.

Low cholesterol is associated with depression among US household population.

The Pharmacology of Lysergic Acid Diethylamide: A Review

#sugarispsychedelic
#serotoninsorrow

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