Whatever Weren’t We Thinking?
The idea that it makes sense to treat physical and psychological symptoms of disease, as though they are separate or unrelated issues, is – although potentially highly profitable – most likely not the best approach to improving long term metabolic health and well-being.
When perceived through a wide lens, it’s not that difficult to see that mood and anxiety disorders, such as chronic depression or generalized anxiety (as well as even schizophrenia and bipolar), go hand in hand with a variety of physical symptoms, or biochemical, metabolic conditions or diseases, and this is not just a coincidence.
In spite of this, metabolic issues – like for instance digestive distress, ongoing low level inflammation, oxidative stress, blood sugar dysregulation and hypothyroidism – continue to be treated as though they are largely inconsequential to, rather than potentially inseparable from the progression of mood related illness.
The problem is, if you’ve been taught to switch off your thinking, and not even begin the process of considering the connections between all the different mental and physical symptoms of disease, it becomes far less likely you will notice them, and even less likely that you will understand them even if you do.
But regardless, the truth of the matter is that psychological dysfunction or instability is directly connected to metabolic performance, and this is reasonably easy to observe, in both directions, with the proper use of the scientific method.
Which is not to suggest that emotional or spiritual matters are unimportant, but rather that things that can interfere with metabolism can also have an impact upon the mind, and things that effect psychological state, also effect the way the body functions. Knowing this can open the way for innovative treatment of diseases of all kinds.
“…5HT [serotonin] release after stress showed a significant increase with helpless behavior…”
Unfortunately, the often contradictory nature of ‘official’ methods of dealing with issues of mind and body, is fueled by a variety of entrenched belief systems, which interfere with the ability to see the natural simplicity of proper metabolic function, precluding many logical and predictably effective solutions.
There is very little good reason to think that the treatment of physical symptoms needs to make psychological symptoms worse, and vice versa. In fact the opposite sounds like a far more reasonable premise.
Unfortunately, a great deal of modern, so called established science, relating to nutrition and treatment, is the product of decades worth of politically influenced, industry driven propaganda, often backed up by biased and poorly performed studies, as well as sometimes fraudulently manipulated results.
Erroneous foundational theories regarding health and disease can be so deeply-rooted, they can be almost invisible at times. But whether or not they are perceived, they are able to powerfully influence decisions and outcomes. Unless and until false premises are brought to light, there is often little realization that they played any part at all.
Ambiguous prescriptions like ‘all things in moderation’, ‘have a balanced diet’, ‘eat your vegetables’, ‘follow a clean eating meal plan’ or ‘consume lots of healthy high-fiber foods’, although common, are misleading, are generally empty of any truly useful information, and are mostly based on incorrect interpretations with regards to nutritional science.
The moderation doctrine can over time, easily move a person into the realm of calorie restriction and under-fueling, when what is often actually needed is better quality fuel and more of it.
The idea of simply eating a balanced diet in order to improve health no longer seems so useful, upon closer examination of the underlying scientific theories behind reasons given for recommending the types and quantities of foods making up the balance.
High fiber, vegetable filled, so called clean diets are anything but clean, relying upon poor quality ‘science’, that completely ignores the digestion interfering, stress promoting, bacteria feeding, inflammatory effects of excessive intake of anti-metabolic substances found in many kinds of veggies, particularly when eaten raw.
It becomes even more difficult to discover the serious harm that can be done by these kinds of notions, because of the existence of vast amounts of poor quality studies, which attempt to frame all sorts of biochemical substances which rise under stress, as being responsible for improving metabolic health.
A good example of this is serotonin, which is often inappropriately referred to as the ‘happiness hormone’, with the implication being that psychological dysfunction is the result of insufficient serotonin availability.
More and more scientific experiments however, are finding what has been scientifically demonstrated many times, over many years, that serotonin is a promoter of symptoms of depression and anxiety and mood dysregulation in general.
The idea that rising serotonin improves mind states, makes even less sense in light of the well understood physiological relationship between high serotonin and many kinds of metabolic issues, like digestive distress, chronic inflammation, as well as the promotion of degenerative disease states including obesity and cancer.
Add to that an awareness of the relationship between increased bacterial endotoxin and rising serotonin (and other stress substances such as nitric oxide and estrogen), in the context of the direct connection between digestion and psychological state, and the idea that depression or anxiety is a low serotonin condition holds little water.
Even though the proposition that there is a causal connection between rising serotonin and the development of learned helplessness (or mood disorder symptoms in general) is not new, this information has done little to interfere with the sale of profitable pharmaceutical products specifically intended to increase 5-HT.
There is no good reason that I know of to believe that more of a substance that directly suppresses metabolism (including brain metabolism) would be necessary for improving psychological issues, unless you also believe that a ‘deficiency’ is bad for metabolism in general. In which case you have to accept that suppressing metabolism is the way to fix health issues. There are people who suggest this, however I am yet to hear an argument that sounds logical and convincing. The fact that serotonin has been shown to promote brain tumors should be enough information alone.
When drugs fail to have their desired effect – or even when they cause a significant worsening of symptoms known to result from chronic stress and serotonin issues – results tend to be disregarded, or rationalized away as unrelated ‘side-effects’.
Biased and inaccurate observation and reporting of treatment results and effects, fuels a murkiness regarding interrelated problems, making the possibility of using logical investigation and holistic understanding to solve numerous complaints simultaneously, very difficult.
It’s little wonder confusion arises when consumption of ‘health foods’ – promoted as healthy because they raise serotonin and nitric oxide, or are high in harmful polyunsaturated fats – cause obvious digestive distress and other symptoms, and worsen anxiety and depressive feelings?
The chances of believing something that makes you feel sick is helping you, go up if there is a disconnect between biology, and popularly promoted approaches to health and well-being.
A more comprehensive look at the history of physiology can make it easier to see the interrelationship between symptoms that, more and more today, are being treated as if they are isolated, unrelated conditions.
When the biochemistry of stress is discussed honestly, logically and holistically, many interactions which are often misunderstood or ignored – like the impact of excess estrogen or serotonin on stress and disease; the relationship between sugar or salt consumption and mood regulation; the inflammatory effects of the breakdown of PUFAs and metabolic illness in general – can begin to be seen as important, interrelated clues for the improvement of metabolism as a whole.
It makes sense that a better understanding of metabolic function would show how things work together more – rather than less – logically and predictably.
Unfortunately, whilst the objectives of big industry and big government remain the driving force behind decisions over what to produce, and to promote, it’s unlikely that genuine biological principles relating to nutrition and disease (pointing to common approaches to a multitude of health related problems) will play a large part.
Although I’m not a doctor or scientist, and none of this is intended as advice, I believe that as long as misinformation and confusion continues to be the driving force behind what is and isn’t considered to be safe or beneficial, it’s unlikely that official approaches to the treatment and prevention of disease (both physical and psychological), will include good information for improved holistic whole metabolism outcomes.
Many still believe that ‘they’re searching for a cure’, and that ‘they have our best interests at heart’, but what if that’s just another product of conditioned, unexamined thought bubbles.
See More Here
Serotonin States and Social Anxiety
Indigenous Bacteria from the Gut Microbiota Regulate Host Serotonin Biosynthesis
Executive functioning and diabetes: The role of anxious arousal and inflammation.
High intelligence: A risk factor for psychological and physiological overexcitabilities
In vivo serotonin release and learned helplessness.
Rapid recovery from major depression using magnesium treatment.
Serotonin content of foods: effect on urinary excretion of 5-hydroxyindoleacetic acid.
A randomised trial of ondansetron for the treatment of irritable bowel syndrome with diarrhoea
Lysine as a serotonin receptor antagonist: using the diet to modulate gut function.
Antinutritional properties of plant lectins.
Straight talk about high-fructose corn syrup: what it is and what it ain’t
Cyproheptadine Use in Children With Functional Gastrointestinal Disorders.
Low Calorie Dieting Increases Cortisol
Increased plasma nitric oxide metabolites in suicide attempters.
Serotonin engages an anxiety and fear-promoting circuit in the extended amygdala
Serotonergic Regulation of Excitability of Principal Cells of the Dorsal Cochlear Nucleus
Whole-Body Hyperthermia for the Treatment of Major Depressive Disorder
Anti-nutritional and toxic factors in food legumes: a review.
Relationship between dietary fiber and cancer: metabolic, physiologic, and cellular mechanisms.
Activation of Meningeal 5-HT2B Receptors: An Early Step in the Generation of Migraine Headache?
HPA axis dysregulation in men with hypersexual disorder.
Insulin resistance in brain alters dopamine turnover and causes behavioral disorders
Treatment of depression with cyproheptadine.
Estrogen-mediated effects on depression and memory formation in females
Psychoregulating role of nicotinamide.
Preventive effect of cyproheptadine hydrochloride in refractory patients with frequent migraine
Bivalent role of intra-platelet serotonin in liver regeneration and tumor recurrence in humans.
Mice Genetically Depleted of Brain Serotonin Do Not Display a Depression-like Behavioral Phenotype
Reducing peripheral serotonin turns up the heat in brown fat.
Selective Serotonin Reuptake Inhibitors and Violent Crime: A Cohort Study
Immunohistochemical study of serotonin in lesions of chronic eczema.
Excessive Sugar Consumption May Be a Difficult Habit to Break: A View From the Brain and Body
Why May Allopregnanolone Help Alleviate Loneliness?
Efficacy of cyproheptadine for nightmares associated with posttraumatic stress disorder.
High-Fat Diet Changes Fungal Microbiomes and Interkingdom Relationships in the Murine Gut
Intestinal fungi contribute to development of alcoholic liver disease
Placental Source for 5-HT that Tunes Fetal Brain Development
An antidepressant that extends lifespan in adult Caenorhabditis elegans.
Serotonin Activated Hepatic Stellate Cells Contribute to Sex Disparity in Hepatocellular Carcinoma
Stimulating healthy tissue regeneration by targeting the 5-HT2B receptor in chronic liver disease
Increased estrogen level can be associated with depression in males.
Serotonin and human cancer: A critical view.
#serotoninsorrow
#estrogeneration
#sugarsaves
#raypeat
Image: yogajournal.ru/body/practice/home-practice/ya-plakal-top-10-samykh-smeshnykh-yoga-kartinok/Jun