La Conga Dulce

Congo Exposure to stress (of any kind) depletes glycogen stores in your body at an increased rate and, as such, can create an immediate need for sugar.

When glycogen in storage begins to run low – and if enough sugar does not get eaten – one way your body can compensate is by releasing the stress hormones adrenaline and cortisol.

Cortisol is used in part by the body to break down muscle tissue into amino acids for conversion into glucose.

Whenever muscle tissue is broken down (or large amounts of pure muscle meat get consumed), higher doses of the inflammatory amino acids – tryptophan, cysteine, methionine, and histidine – get released into circulation in the blood.

Increased free circulation of the above amino acids can, directly and indirectly, lead to the suppression of thyroid energy system metabolism and interference with immune system function.

Tryptophan is the precursor to serotonin – thus, consumption and increased release into circulation cause serotonin levels to rise – and this is another factor in the suppression of thyroid and immune system function, as well as the promotion of inflammatory disease.

Histidine  – the precursor to histamine – when circulating in increasing quantities, can also be a significant factor concerning the advancement of issues of inflammation and degeneration.

Adrenaline encourages the release of free fatty acids into the bloodstream, which – mainly when polyunsaturated – can cause the further release of cortisol (and adrenaline), helping to create a vicious circle of stress and promote increased rates of ageing, inflammation and disease.

It is crucial to remember that continuous exposure to stress – and thyroid suppression in general – can often be enough to trigger issues related to inflammation and immune system dysfunction. However, it is equally valid in reverse, as none of these states can be independent.

Consuming sugar from ripe sweet fruits, fruit juice, milk, honey, and white sugar – and to a lesser extent, well-cooked starchy vegetables like a potato – can have a powerfully protective anti-stress effect.

Increasing their intake (and avoiding the anti-metabolic ingredients) is a safe and reasonable approach to improving a wide variety of disease states, which, one way or another, are related to excessive inflammation, interference with immune system function, and metabolic under-performance in general.

Supplementing with gelatin – or choosing the more gelatinous cuts of meat – is said to help protect against inflammation, providing far less problematic amino acids and more protective anti-inflammatory amino acids, in particular glycine.

In some ways, glycine (or gelatin more generally) – much like sugar – can be seen as an antidote to the stress-promoting effects of the excess circulation of the inflammatory amino acids. So it is regardless of whether levels are rising due to the consumption of muscle meats alone. Or as a result of increased cortisol levels (due to stress or sugar restriction), or a combination of both.

When discussing the damaging, disease-promoting effects of stress, few combinations could be worse (as far as diet is concerned) than the low sugar, high polyunsaturated fat, increased muscle meat diet, popularly promoted as healthy in many circles today.

You can’t talk about the impact of stress, inflammation and immune system dysfunction without mentioning how metabolic suppression and disease connect to digestion issues and excessive exposure to bacteria and endotoxin.

A lack of sugar, too much exposure to polyunsaturated fats, and an inflammatory balance of circulating amino acids are enough to drive the gradual worsening of digestive issues. But limiting the intake of grains, beans, legumes, and other starchy and fibrous materials, including undercooked vegetable matter, can be helpful.

I’m not a doctor or health professional, and this is not advice. But experimentation with different combinations of the above ideas is believed by some to be an appropriate and potentially effective means to protection – and recovery – from inflammatory disease and immune system interference in general, improving overall resilience to stress.

Copyright 2021, by Dan M @ CowsEatGrass. All rights reserved (except for quotations and images having their own protected copyrights). This copyright protects author-publisher Dan M’s right to future publication of his work in any manner, in any and all media — utilizing technology now known or hereafter devised — throughout the world in perpetuity. Everything described in this publication is for information purposes only. The author-publisher, Dan M, is not directly or indirectly presenting or recommending any part of this publication’s data as a diagnosis or prescription for any ailment of any reader. If anyone uses this information without the advice of their professional health adviser, they are prescribing for themselves, and the author- publisher assumes no responsibility or liability. Persons using any of this data do so at their own risk and must take personal responsibility for what they don’t know as well as for what they do know.

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Carvalho DP, Ferreira AC, Coelho SM, Moraes JM, Camacho MA, Rosenthal D. Thyroid peroxidase activity is inhibited by amino acids. Braz J Med Biol Res. 2000 Mar;33(3):355-61.

Laugero KD, Bell ME, Bhatnagar S, Soriano L, Dallman MF. Sucrose ingestion normalizes central expression of corticotropin-releasing-factor messenger ribonucleic acid and energy balance in adrenalectomized rats: a glucocorticoid-metabolic-brain axis? Endocrinology. 2001 Jul;142(7):2796-804.

Pecoraro N, Reyes F, Gomez F, Bhargava A, Dallman MF. Chronic stress promotes palatable feeding, which reduces signs of stress: feedforward and feedback effects of chronic stress. Endocrinology. 2004 Aug;145(8):3754-62.

Patrice D. Cani, Rodrigo Bibiloni, Claude Knauf, Aurélie Waget, Audrey M. Neyrinck, Nathalie M. Delzenne, Rémy Burcelin; Changes in Gut Microbiota Control Metabolic Endotoxemia-Induced Inflammation in High-Fat Diet–Induced Obesity and Diabetes in Mice. Diabetes 1 June 2008; 57 (6): 1470–1481.

Zhong Z, Wheeler MD, Li X, Froh M, Schemmer P, Yin M, Bunzendaul H, Bradford B, Lemasters JJ. L-Glycine: a novel antiinflammatory, immunomodulatory, and cytoprotective agent. Curr Opin Clin Nutr Metab Care. 2003 Mar;6(2):229-40.



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